Intracellular calcium increase in epileptiform activity: modulation by levetiracetam and lamotrigine
Articolo
Data di Pubblicazione:
2004
Abstract:
Alterations in neuronal calcium (Ca2+) homeostasis are believed to play an essential role in the generation and propagation of epileptiform events. Levetiracetam (LEV) and lamotrigine (LTG), novel antiepileptic drugs (AEDs), were tested on epileptiform events and the corresponding elevations in intracellular Ca2+ concentration ([Ca2+]i) recorded from rat neocortical slices.
Tipologia CRIS:
1.1 Articolo in rivista
Keywords:
Animals; Pyramidal Cells; Calcium; Dose-Response Relationship; Drug; Triazines; Bicuculline; Calcium Channel Blockers; Calcium Channels; N-Type; Rats; Calcium Channels; Patch-Clamp Techniques; Epilepsy; Rats; Wistar; Piracetam; Membrane Potentials; 4-Aminopyridine; Anticonvulsants; Male; Neocortex
Elenco autori:
Pisani, Antonio; Bonsi, P; Martella, Giuseppina; De Persis, C; Costa, C; Pisani, F; Bernardi, Giorgio; Calabresi, Paolo
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